Questions from small group to help prepare for Course 3 final exam
Assess ABCs
Procainamide
Magnesium
High-energy defibrillation
Apply CPR
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HR very rapid and the rhythm is not regular
QRS complex is absent
P waves are absent
Wide QRS complexes
Voltage fluctuations ("coarse" and "fine")
Can look like asystole
No relationship between P waves and QRS complexes
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Resume CPR immediately after electrical defibrillation. Continue for 5 cycles of CPR, then assess and continue the treatment algorithm. Three stacked shocks are no longer recommended
Do not allow the chest to fully recoil between compressions.
CPR should be performed until the defibrillator is available. Effective CPR is most important when the first defibrillation attempt is made more than four minutes after the arrest
For adults that are not intubated, compress 30 times then deliver 2 positive pressure breaths. Repeat this cycle at 100 compressions per minute.
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Early electrical defibrillation is the definitive treatment for V fib
Defibrillate as soon as possible. For an unwitnessed arrest outside the hospital, there is some evidence that electrical defibrillation may be more effective after 5 cycles of CPR.
The initial energy setting should be 120-200 Joules for biphasic defibrillators and 200-360 Joules for monophasic defibrillators. Set to 200 Joules if the defibrillator is not clearly labeled as monophasic or biphasic.
Resume CPR immediately after electrical defibrillation. Continue for 5 cycles of CPR, then assess and continue the treatment algorithm. Three stacked shocks are no longer recommended.
Subsequent shock energy should be equal to or lesser than previous shock energy
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The optimum sternal electrode position is just to the left of the upper sternal border below the clavicle.
The optimum sternal electrode position is just to the right of the upper sternal border below the clavicle.
The optimum apex position is to the left of the left nipple with the center of the electrode in the midaxillary line.
Anteriorly and posteriorly over the left hemithorax
Beneficial mainly due to alpha stimulation, which increases systemic vascular resistance and improves coronary and cerebral blood flow.
Beneficial mainly due to alpha stimulation, which increases myocardial electrical activity and strength of contraction
Beneficial mainly due to beta stimulation, which increases systemic vascular resistance and improves coronary and cerebral blood flow
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Cardiac arrest from v fib
Cardiac arrest from VT
Reverse the effect of beta-blockers
Cardiac arrest from asystole
Extreme hypotension
Cardiac arrest from pulseless electrical activity
Bradycardia
Heart block
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1 mg IV push every 3-5 min
2-5 mg IV push every 3-5 min
1 mg, 3 mg, 5 mg IV push 3 min apart
2-2.5 mg per ETT via endotracheal tube
All of the above
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1 mg IV push every 3-5 min.
1 mg, 3 mg, 5 mg IV push 3 min apart
2-10 mcg/min continuous infusion
All of the above
Hypertension
Tachycardia
Arrhythmias
Myocardial ischemia
Stroke
Absence of a detectable pulse with electrical activity other than VT or V fib
Asystole
VT or V fib
Hypovolemia
Hypoxia
Hydrogen ion-acidosis
Hyper/hypokalemia
Hypothermia
Myocardial Infarction
Tablets (drugs)
Tamponade-cardiac
Tension pneumothorax
Thrombosis - coronary
Thrombosis - pulmonary
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ABCs
Vigorous chest compressions
Fluid boluses
Epinephrine to improve coronary perfusion
Amiodarone if bradycardic
Determine underlying etiology
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Confusion
Lethargy
Chest pain
Sepsis
Severe hypotension
Pulmonary edema
Shortness of breath
Wide QRS
Narrow QRS
AV dissociation
Regular rhythm
Fusion or capture beat
NW axis
Positive QRS concordance in anterior leads
Begin CPR before preparing for cardioversion
Treat pulseless VT the same as V fib
Unsynchronized cardioversion
Start cardioversion at 360 joules
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Heart rate slow to normal
Rhythm not regular - some QRS complexes missing
Wide QRS
P waves are upright
There are more P waves than QRS complexes.
The PR interval progressively lengthens
The shortest PR interval follows the dropped beat.
The PR interval progressively lengthens until a QRS complex is dropped.
Inferior wall infarctions
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Heart rate slow to normal, irregular rhythm
QRS complexes missing
More P waves than QRS complexes
Fixed PR interval
Inferior wall infarctions
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Heart rate slow to normal, irregular rhythm
Wide QRS
More P waves than QRS complexes
Atrial and ventricular regularity independent of each other
P waves "march through" QRS complexes
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Chest pain
Syncope
Severe dyspnea
Impending sense of doom
Arrhythmia
Decreased level of consciousness
Hypotension
Congestive heart failure
Atropine 0.5-1.0 mg IV, repeat every 3-5 minutes
Transvenous pacemaker
Dopamine 2-10 mcg/kg/min
Epinephrine 2-10 mcg/min
Transcutaneous pacemaker
None of the above
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Parasympathetic - reverses bradycardia due to excessively high vagal tone
Competitive blockade of acetylcholine at muscarinic receptors
Increases sinus node automaticity and AV conduction
Increases bronchial secretions
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Tachyarrhythmias
Exacerbation of MI
Paradoxical bradycardia
Dry mouth, urinary retention, blurred vision, flushed skin
Ataxia, delirium, coma
Hematoma
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