Medications are put in place to either ease a patient’s pain or to revert them to health. Being that you are a pharmacology student, the one thing that you should do is ensure you know which types of drugs to prescribe and when. Take up the quiz below and get to see how much you know about respiratory and gastrointestinal medication.
Reducing stress
Avoiding aggrevating foods
Quit smoking
A 4 oz glass of red wine 3x/week
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Aspirin
Acetaminophen
Diphenhydramine (Benadryl)
Warfarin
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Aspirin
Acetaminophen
Ibuprophen
Naproxen
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Binding to CCK2 (cholecystekinin2) receptors to activate protein kinases to secrete H+ ions
Causing vasodilation and increased release of HCL from the islet cells
Inhibiting platelet aggregation whereby decreasing competition for H+ binding sites
Binding with H+ ions to create viscous coating of epithelial lining of the stomach
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Bropheneramine (Veltane)
Indomethacin (Indocin)
Naproxen
Fenamic acid
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Dextromethorophan
Hydrocodone
Celecoxib (Celebrex)
Meloxicam (Mobic)
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Aspirin
Acetaminophen
Nalbumetone (Relafin)
Ibuprofen
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Weakly inhibits cylcooxygenase
Metabolized in the liver via glucoronidation
Analgesic, antipyretic and anit-inflammatory effects
Interacts with Coumadin
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1-2 days
1-2 weeks
5-7 days
6-8 weeks
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High albumin binding
May decrease effects of ACE inhibitors
Reversibly inhibits thromboxane
Absorption is decreased with higher pH's
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Provide analgesia at 50% of the anti-inflammatory dose
Reversibly inhibit the COX system
Are typically only well absorbed in the duodenem
Are highly plasma-protein binding
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Increases gastrin production
Decreases blood flow
Decreases healing
Decreases HCO3 production
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Vasodilation
Platelet aggregation
Gastric cytoprotection
Pain sensitization
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Pseudoephedrine
Oxymetazoline
Tetrahydrazoline
Desloratadine (Clarinex)
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Pancreatic cancer
CHF
Wilm's tumor
HTN
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Present in low amounts in kidney, platelets and GI tract
Involved in thromboxane synthesis
Induced by cytokines
Renal vasodialtion
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