Questions about the things mentioned in the title. . . . Contains quite a few questions related to the AMK and is linked to the "Old Age 1" case unit.
Isodosis
Acidosis
Alkalosis
Alkalaemia
Acidaemia
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Controlling osmosis between compartments
Maintaining acid-base balance
Carry electrical currents
Serve as cofactors
All of the above
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Aldosterone
Insulin
Renin
Krebs cycle
Gucagon
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The main regulators of magnesium in the blood
The main regulators of phosphate in the blood
The main regulators of calcium in the blood
The main regulators of NaCl in the blood
The main regulators of bicarbonate in the blood
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ANP
Angiotensin II
Renin
ADH
Angiotensinogen
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Sodium
Chloride
Potassium
Phosphate
Bicarbonate
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Antidiuretic hormone
ANP
Aldosterone
Parathyroid hormone
ACE
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Plasma
Cytosol
Interstitial fluid
Lymph
Bile
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Skin
Water
Blood
Organs
Electrolytes
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Sodium
Chloride
Potassium
Phosphate
Bicarbonate
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10%
50%
33%
66%
99%
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Ways to balance interstitial fluid
Means of balancing blood volume
Ways to eliminate H+ from the body
Ways to make ions
Ways to increase blood volume
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Antidiuretic hormone
ANP
Aldosterone
Parathyroid hormone
Renin
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Haemorrhage
Vomiting
Kidney failure
Pneumothorax
Diabetes
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Lipids
Plasma membranes
DNA
Calcium phosphate salt
Carbohydrates
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Anions
Cations
Metabolic water
Lipids
Carbohydrates
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PH is brought into the normal range
Systemic arterial pH is still lower than 7.35
Systemic arterial pH is still higher than 9.5
PH is greater than 5.5
PH is lower than 2.5
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Dilute body fluids
Decrease in the osmolarity of interstitial fluids
Osmosis of water from ICF to ECF
Dilute body fluids and a decrease in the osmolarity of interstitial fluids
Decrease in water intake
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Sodium
Chloride
Potassium
Phosphate
Bicarbonate
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Metabolic alkalosis
Meteabolic acidosis
Respiratory acidosis
Respiratory alkalosis
Combined respiratory and metabolic alkalosis
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Blood pH to drop
Alkalosis
Respiratory compensation
Metabolic acidosis
Metabolic alkalosis
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Sodium
Calcium
Magnesium
Phosphate
Manganese
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Metabolic alkalosis
Metabolic acidosis
Respiratory acidosis
Respiratory alkalosis
Combined respiratory and metabolic alkalosis
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Bradycardia
Hypotension
Tachycardia
Confusion
Muscular weakness
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Dehydration and hypernataemia due to inadequate fluid intake or loss of more water than sodium in vomit, faeces, or urine
Hyponatraemia due to inadequate intake of sodium, impaired kidney function, or excessive sodium loss
Hypokalaemia due to excessive laxative use or potassium-depleting diuretics
Hyperkalaemia due to increased dietary intake
Acidosis due to lung or kidney disease
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Decreased Na+ levels in urine
Decreased Cl- levels in urine
Increased K+ levels in urine
Increased PO- levels in urine
None of the above
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Dehydration
Formation of angiotensin II
Stimulate the kidneys to secrete renin
Formation of ADH
Increased vasoconstriction
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Sodium
Chloride
Potassium
Phosphate
Bicarbonate
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Thirst centre
Fluid balance
Urinary salt loss
Compartmentalizing
Perspiration
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Is twice as fast as an adult's
Causes greater water loss from the lungs
Removes less carbon dioxide than in an adult
Makes the infant blood more acidic
Is the same as an adult's
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Chloride shift
Potassium shift
Sodium shift
Bicarbonate shift
Protein shift
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They are equal
2 times higher
5 times higher
7 times higher
10 times higher
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Increased GFR
Reduced Na+ and Cl- reabsorption by the kidneys
Reduced water reabsorption by the kidneys
Increased calcium reabsorption
Increased urine output
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Hydrogen ions
Bicarbonate ions
Nonvolatile acids
Volatie bases
Calcium ions
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