The quiz will assess the nurse's knowledge of cardiac drips.
Dose should be omitted only for that day.
Client needs a dose of potassium before receiving digoxin.
Dose should be withheld and the healthcare provider notified.
Dose should be administered as ordered
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Call Code Blue
Begin CPR
Defibrillate immediately at 360 joules (monophasic) or 200 joules (biphasic)
Check monitor to verify whether rhythm changes
All the above.
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HCT and HGB
Drowsiness
Decreased renal function
Loss of appetite
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Maligant ventricular ectopy. (frequent PVC's, Vtach, Vfib.
Unstable or chronic angina in hemodynamically stable patient.
Atrial flutter or Atrial Fibrillation with rapid ventricular response.
Decreased cardiac output.
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Obtain 12 lead EKG
Attempt to slow patient's heart rate through use of vagal maneuvers. Ask patient to cough and bear down.
Call the physician.
Give Adenosine 6 mg rapid IV push over 1-3 seconds, with rhythm strip running continuously.
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Titrate by 10 mcg/min every five minutes.
Titrate by 10 mcg/min every 15 minutes.
Titrate by 5 mcg/min every 5 minutes.
Do not titrate, call physician for orders.
Depresses excitability of cardiac muscle, slows conduction to atrium, bundle of HIS and ventricles.
Causes vasodilation. Mild diruetic effect.
Delay in rate of repolarization, prolonged action potential phase, slowing of the speed of electrical conduction through normal and accessory pathways.
Increases electrical stimulation threshold of ventricle, stabilizes cell membrane, decreases automaticity.
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Continue as previously checked.
Be repeated as indicated under "initial monitoring".
Be checked every 8 hours.
Every 30 minutes/2 hours.
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Call Physician and monitor patient.
Give Adenosine 6 mg rapid IV push over 1-3 seconds, with rhythm strip running continuously.
Call Code Blue and begin CPR.
Administer Lidocaine bolus 1-1.5 mg/kg then start Lidocaine drip.
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Increases electrical stimulation threshold of ventricle, stabilizes cell membrane, decreases automaticity
Causes vasodilation. Mild diruetic effect.
Delay in rate of repolarization, prolonged action potential phase, slowing of the speed of electrical conduction through normal and accessory pathways.
Depresses excitability of cardiac muscle, slows conduction to atrium, bundle of HIS and ventricles
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