Take this quiz and prove yourself now, how well do you know about heart disease?
Cardiac output is determined by the heart rate and pulse
Stimulation of the sympathetic nerves decreases cardiac output
Vagal discharge increases cardiac output
Cardiac output is determined by heart rate and stroke volume
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Parasympathetic
Slows heart rate
(+) inotropic effect
Autonomic
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(-) inotropic effect
Increased heart rate
Increased stroke volume
Increased cardiac output
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Heart rate
Preload
End systolic volume
Blood pressure
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(+) inotropic effect
(+) dromotropic effect
Starling's law of the heart
(-) chronotropic effect
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Mm Hg
Ml/min
Ml/beat
Liters/24 hr
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Blood is ejected from the ventricles
The semilunar valves are open
The atrioventricular valves are closed
Blood fills the ventricles
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Increased heart rate
Increased stroke volume
Increased venous return (Starling's law of the heart)
Vagal discharge
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Beta1 adrenergic receptor activation
Parasympatholytic effect
Vagal discharge
Muscarinic blockade
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Stroke volume
Heart rate
Pulse
Cardiac output
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Increases heart rate
Causes bradycardia
Is also classified as sympathomimetic
Is vagomimetic
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End-systolic volume
Stroke volume
Preload
Ejection fraction
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Sympathetic nervous systemactivation
Beta1 adrenergic receptor activation
(+) dromotropic effect
Vagal discharge
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Less than 60 beats/min
Vagal discharge
(-) chronotropic effect
Sympathetic nerve stimulation
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Increases afterload
Decreases preload
Increases end diastolic volume
Stretches the ventricular myocardium
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Blocks the beta 1 adrenergic receptors and causes a (-) chronotropic and (-) inotropic effect
Activates the beta1 adrenergic receptors and causes bronchoconstriction
Activates the beta1 adrenergic receptors and causes a (+) inotropic effect
Activates the vagus nerve, thereby slowing heart rate and strengthening myocardial contraction
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Tachycardia
Low blood pressure
Ankle edema
Slow heart rate
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Ventricular depolarization
The opening of the valves of the ventricles
Ventricular filling
Contraction of the ventricular myocardium
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Exercise
Sympathetic nerve stimulation
(+) inotropic response
Heart failure
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