Explore the Neuromuscular Junction (NMJ), focusing on its function, the role of ACh, nicotinic receptors, and neuromuscular blocking agents. This quiz assesses understanding of synaptic interactions and pharmacological interventions in neuromuscular transmission.
Pancuronium
Succinylcholine
Vecuronium
Rocuronium
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Depolarization effect on the postsynaptic muscle is long lasting
A short period of repetitive excitation and muscle twitching followed by…Blockade of neurotransmission and paralysis
Initial action of depolarizing agents is to cause opening of ion channels
All are true
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Muscarinic receptors
Nicotinic receptors
Dopamine receptors
Serotonin receptors
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Polarizing agents
Depolarizing agents
Competitive agents
Noncompetitive agents
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Polarizing agents
Depolarizing agents
Competitive agents
Noncompetitive agents
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Histamine, long
5-HT, long
Histamine, short
5-HT, short
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True
False
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Has actions on both effector and chemosensory sites
Can rapidly cause receptor desensitization
Causes a transient stimulation of all autonomic ganglia
Ultimate response is due to summation of all nicotine’s effects on an organ/system
Pharmacological actions are complex, unpredictable
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Natural alkaloids and congeners
Ammonio steroids
Benzylisquinolones
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Prototype ammonio steroid
No histamine release
Block muscarinic receptors, and causes tachycardia
Don’t have ganglionic blocking actions, don’t alter vagus nerve function
Prototype benzylisquinolone
Results in bronchospasm, hypotension, increased secretory activity
Can cause histamine release
Dangerous in patients with CHF who are taking diuretics, digoxin
Life threatening in individuals with ion regulation dysfunction
Dangerous in patients withsignificant burns, soft tissue damage, and in children
Cause a significant release of K+ from intracellular sites
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Nicotinic Neurotransmission
Muscarinic Neurotransmission
Ganglionic Neurotransmission
Cholinergic Neurotransmission
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Edrophonium
Neostigmine
Pyridostigmine
Atropine
Trimethaphan
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Prototype ammonio steroid
No histamine release
Block muscarinic receptors, and causes tachycardia
Don’t have ganglionic blocking actions, don’t alter vagus nerve function
Prototype benzylisquinolone
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Postsynaptic cell
K+ is depleted
Butyrylcholinesterases
Presynaptic cell
K+ is increased
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Prototype ammonio steroid
No histamine release
Block muscarinic receptors, and causes tachycardia
Don’t have ganglionic blocking actions, don’t alter vagus nerve function
Prototype benzylisquinolone
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